Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the flip side, our power to rescue SERT functionality in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream in the Pro32Pro33 mutant, as confirmed by amplified pFAK localization in https://cicerob108hrc0.wiki-jp.com/user